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Question #45722
The Question
In a clinical scenario, a patient possesses a genetic mutation that renders the receptors for Atrial Natriuretic Factor (ANF) completely insensitive. During an episode of increased venous return and high blood volume, which of the following physiological consequences will be observed?
Options
- A.
The renin-angiotensin mechanism will remain unchecked, leading to persistent sodium retention and hypertension.
- B.
Systemic vasodilation will eventually occur due to the massive accumulation of unbound ANF in the blood.
- C.
The Juxtaglomerular (JG) cells will directly sense the atrial stretch and independently inhibit renin release.
- D.
Blood pressure will rapidly decrease due to a compensatory surge in Anti-Diuretic Hormone (ADH) secretion.
Check Your Answer
Correct Answer
Step-by-Step Solution
Explanation
How the Examiner Thinks
Regulatory_Failure
Understand how NOT, EXCEPT, and logical constraints are used to manipulate exam results.
Concepts Behind This Question
Renin|Angiotensin II|Aldosterone|ANF
Why This Question Is Tricky
Common Mistakes
Conceptual Pivot: Recognizing ANF as the obligatory endocrine 'brake' on the RAAS pathway.
A common misconception is that high blood volume mechanically shuts down RAAS entirely on its own, or that high circulating hormone levels (Option B) can overcome receptor absence.
This tests the student's understanding of receptor-mediated endocrine antagonism.
Quick Facts About This Question
Question Information
| Subject | Biology |
| Chapter | Excretory Products and Their Elimination - Class 11 |
| Concept | RAAS Pathway Feedback Loop |
| Difficulty | 8 |
| Cognitive Load | 5 |
| Target Solve Time | 120 sec |
| Access | Free |