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Question #47429
The Question
The fetal ejection reflex relies on a synergistic neuroendocrine mechanism involving maternal oxytocin and local prostaglandins. If a full-term pregnant female is administered a pharmacological agent that inhibits local prostaglandin synthesis in the uterine tissue, which of the following will be the most direct physiological consequence?
Options
- A.
The maternal neurohypophysis will completely cease oxytocin secretion due to the loss of the primary afferent neural stimulus.
- B.
The myometrium will fail to develop sufficient gap junctions, resulting in uncoordinated and weak uterine contractions despite the presence of oxytocin.
- C.
The fetal adrenal cortex will be inhibited from secreting cortisol, preventing the initiation of the fetal ejection reflex.
- D.
The placenta will immediately halt the secretion of relaxin, preventing the necessary widening of the pubic symphysis.
Check Your Answer
Correct Answer
Step-by-Step Solution
Explanation
How the Examiner Thinks
Comparative_Analysis
Sharpen your ability to evaluate multiple scenarios and distinguish between competing potential answers.
Concepts Behind This Question
Hypothalamus|Adenohypophysis|Neurohypophysis|Uterine_Contraction
Why This Question Is Tricky
Common Mistakes
Conceptual Pivot: Students often memorize oxytocin as the sole effector of uterine contractions (the 'hormonal' component) while neglecting the local paracrine role of prostaglandins.
The trap lies in assuming that as long as oxytocin is present, contractions will be normal.
This question tests the NCERT-Plus understanding that oxytocin requires PG-induced gap junctions to produce the synchronized, forceful contractions necessary for successful parturition.
Quick Facts About This Question
Question Information
| Subject | Biology |
| Chapter | Neural Control and Coordination - Class 11 |
| Concept | Synaptic Vesicle Exocytosis Trigger |
| Difficulty | 8 |
| Cognitive Load | 5 |
| Target Solve Time | 120 sec |
| Access | Free |